Jamie Cameron

     

    Publications 2017

    Roffey S, Tucker K, Filipek-Ogden K, Montgomery J, Cameron J, O'Connell T, Evans J, Marter P, Taylor GM (2017)
    Investigation of a Medieval Pilgrim Burial Excavated from the Leprosarium of St Mary Magdalen Winchester, UK
    PLoS Negl Trop Dis, 11 (1), e0005186
    DOI 10.1371/journal.pntd.0005186, PubMed 28125649

    Publications 2016

    Wierød L, Cameron J, Strøm TB, Leren TP (2016)
    Studies of the autoinhibitory segment comprising residues 31-60 of the prodomain of PCSK9: Possible implications for the mechanism underlying gain-of-function mutations
    Mol Genet Metab Rep, 9, 86-93
    DOI 10.1016/j.ymgmr.2016.11.003, PubMed 27896130

    Publications 2014

    Cameron J (2014)
    New dentist spotlight: Dr. Jamie Cameron
    J Okla Dent Assoc, 105 (1), 27
    PubMed 24624747

    Publications 2012

    Cameron J, Bogsrud MP, Tveten K, Strøm TB, Holven K, Berge KE, Leren TP (2012)
    Serum levels of proprotein convertase subtilisin/kexin type 9 in subjects with familial hypercholesterolemia indicate that proprotein convertase subtilisin/kexin type 9 is cleared from plasma by low-density lipoprotein receptor-independent pathways
    Transl Res, 160 (2), 125-30
    DOI 10.1016/j.trsl.2012.01.010, PubMed 22683370

    Tveten K, Strøm TB, Cameron J, Berge KE, Leren TP (2012)
    Mutations in the SORT1 gene are unlikely to cause autosomal dominant hypercholesterolemia
    Atherosclerosis, 225 (2), 370-5
    DOI 10.1016/j.atherosclerosis.2012.10.026, PubMed 23102784

    Publications 2011

    Holla ØL, Cameron J, Tveten K, Strøm TB, Berge KE, Laerdahl JK, Leren TP (2011)
    Role of the C-terminal domain of PCSK9 in degradation of the LDL receptors
    J Lipid Res, 52 (10), 1787-94
    DOI 10.1194/jlr.M018093, PubMed 21771976

    Holla ØL, Laerdahl JK, Strøm TB, Tveten K, Cameron J, Berge KE, Leren TP (2011)
    Removal of acidic residues of the prodomain of PCSK9 increases its activity towards the LDL receptor
    Biochem Biophys Res Commun, 406 (2), 234-8
    DOI 10.1016/j.bbrc.2011.02.023, PubMed 21324305

    Strøm TB, Tveten K, Holla ØL, Cameron J, Berge KE, Leren TP (2011)
    The cytoplasmic domain is not involved in directing Class 5 mutant LDL receptors to lysosomal degradation
    Biochem Biophys Res Commun, 408 (4), 642-6
    DOI 10.1016/j.bbrc.2011.04.077, PubMed 21531209

    Strøm TB, Tveten K, Holla ØL, Cameron J, Berge KE, Leren TP (2011)
    Characterization of residues in the cytoplasmic domain of the LDL receptor required for exit from the endoplasmic reticulum
    Biochem Biophys Res Commun, 415 (4), 642-5
    DOI 10.1016/j.bbrc.2011.10.127, PubMed 22079632

    Tveten K, Holla ØL, Cameron J, Strøm TB, Berge KE, Laerdahl JK, Leren TP (2011)
    Interaction between the ligand-binding domain of the LDL receptor and the C-terminal domain of PCSK9 is required for PCSK9 to remain bound to the LDL receptor during endosomal acidification
    Hum Mol Genet, 21 (6), 1402-9
    DOI 10.1093/hmg/ddr578, PubMed 22156580

    Tveten K, Strøm TB, Cameron J, Holla ØL, Berge KE, Leren TP (2011)
    Characterization of a naturally occurring degradation product of the LDL receptor
    Mol Genet Metab, 105 (1), 149-54
    DOI 10.1016/j.ymgme.2011.10.008, PubMed 22078455

    Publications 2010

    Strøm TB, Holla ØL, Tveten K, Cameron J, Berge KE, Leren TP (2010)
    Disrupted recycling of the low density lipoprotein receptor by PCSK9 is not mediated by residues of the cytoplasmic domain
    Mol Genet Metab, 101 (1), 76-80
    DOI 10.1016/j.ymgme.2010.05.003, PubMed 20659812

    Publications 2009

    Cameron J, Holla ØL, Kulseth MA, Leren TP, Berge KE (2009)
    Splice-site mutation c.313+1, G>A in intron 3 of the LDL receptor gene results in transcripts with skipping of exon 3 and inclusion of intron 3
    Clin Chim Acta, 403 (1-2), 131-5
    DOI 10.1016/j.cca.2009.02.001, PubMed 19361455

    Cameron J, Ranheim T, Halvorsen B, Kulseth MA, Leren TP, Berge KE (2009)
    Tangier disease caused by compound heterozygosity for ABCA1 mutations R282X and Y1532C
    Atherosclerosis, 209 (1), 163-6
    DOI 10.1016/j.atherosclerosis.2009.08.039, PubMed 19765707

    Holla ØL, Strøm TB, Cameron J, Berge KE, Leren TP (2009)
    A chimeric LDL receptor containing the cytoplasmic domain of the transferrin receptor is degraded by PCSK9
    Mol Genet Metab, 99 (2), 149-56
    DOI 10.1016/j.ymgme.2009.09.012, PubMed 19828345

    Strøm TB, Holla ØL, Cameron J, Berge KE, Leren TP (2009)
    Loss-of-function mutation R46L in the PCSK9 gene has little impact on the levels of total serum cholesterol in familial hypercholesterolemia heterozygotes
    Clin Chim Acta, 411 (3-4), 229-33
    DOI 10.1016/j.cca.2009.11.008, PubMed 19917273

    Publications 2008

    Cameron J, Holla ØL, Berge KE, Kulseth MA, Ranheim T, Leren TP, Laerdahl JK (2008)
    Investigations on the evolutionary conservation of PCSK9 reveal a functionally important protrusion
    FEBS J, 275 (16), 4121-33
    DOI 10.1111/j.1742-4658.2008.06553.x, PubMed 18631360

    Cameron J, Holla ØL, Laerdahl JK, Kulseth MA, Berge KE, Leren TP (2008)
    Mutation S462P in the PCSK9 gene reduces secretion of mutant PCSK9 without affecting the autocatalytic cleavage
    Atherosclerosis, 203 (1), 161-5
    DOI 10.1016/j.atherosclerosis.2008.10.007, PubMed 19022446

    Cameron J, Ranheim T, Kulseth MA, Leren TP, Berge KE (2008)
    Berberine decreases PCSK9 expression in HepG2 cells
    Atherosclerosis, 201 (2), 266-73
    DOI 10.1016/j.atherosclerosis.2008.02.004, PubMed 18355829

    Publications 2007

    Holla ØL, Cameron J, Berge KE, Ranheim T, Leren TP (2007)
    Degradation of the LDL receptors by PCSK9 is not mediated by a secreted protein acted upon by PCSK9 extracellularly
    BMC Cell Biol, 8, 9
    DOI 10.1186/1471-2121-8-9, PubMed 17328821

    Publications 2006

    Cameron J, Holla ØL, Ranheim T, Kulseth MA, Berge KE, Leren TP (2006)
    Effect of mutations in the PCSK9 gene on the cell surface LDL receptors
    Hum Mol Genet, 15 (9), 1551-8
    DOI 10.1093/hmg/ddl077, PubMed 16571601

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